Hepatitis G

Hepatitis G is a newly discovered form of liver inflammation caused by hepatitis G virus (HGV), a distant relative of the hepatitis C virus.

HGV, also called hepatitis GB virus, was first described early in 1996. Little is known about the frequency of HGV infection, the nature of the illness, or how to prevent it. What is known is that transfused blood containing HGV has caused some cases of hepatitis. For this reason, patients with hemophilia and other bleeding conditions who require large amounts of blood or blood products are at risk of hepatitis G. HGV has been identified in between 1-2% of blood donors in the United States. Also at risk are patients with kidney disease who have blood exchange by hemodialysis, and those who inject drugs into their veins. It is possible that an infected mother can pass on the virus to her newborn infant. Sexual transmission also is a possibility.

Often patients with hepatitis G are infected at the same time by the hepatitis B or C virus, or both. In about three of every thousand patients with acute viral hepatitis, HGV is the only virus present. There is some indication that patients with hepatitis G may continue to carry the virus in their blood for many years, and so might be a source of infection in others.

Causes and Symptoms

Some researchers believe that there may be a group of GB viruses, rather than just one. Others remain doubtful that HGV actually causes illness. If it does, the type of acute or chronic (long-lasting) illness that results is not clear. When diagnosed, acute HGV infection has usually been mild and brief. There is no evidence of serious complications, but it is possible that, like other hepatitis viruses, HGV can cause severe liver damage resulting in liver failure. The virus has been identified in as many as 20% of patients with long-lasting viral hepatitis, some of whom also have hepatitis C.


The only method of detecting HGV is a complex and costly DNA test that is not widely available. Efforts are under way, however, to develop a test for the HGV antibody, which is formed in response to invasion by the virus. Once antibody is present, however, the virus itself generally has disappeared, making the test too late to be of use.


Testing for a virus that does not seem to cause any illness is generally confined to research purposes. Antibodies can be tested for in blood and a PCR test can show the presence of the virus.


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